Description
Anadrol Oil is the oily injection form of Oxymetholone. The substance itself is identical with classic oral Anadrol, just in oily vehicle for intramuscular administration. The first pass through the liver is bypassed, reducing hepatotoxicity.
Per study reports pharmacology remains identical: highly-potent androgen receptor agonist with additional direct estrogen receptor activation (paradoxical estrogen effect without aromatization), strong water retention, rapid mass gain. Effects on hematocrit (Anadrol was originally an anemia medication) are the same under injection.
The main advantage of the injectable form: substantially reduced first-pass hepatic load. Classical oral Anadrol is one of the most hepatotoxic anabolics of the class; the injection distributes systemically without the intense liver first-pass.
Pharmacokinetically the oily injection has a slightly longer effective half-life than the tablet (~10-12 hours), making daily injections practicable.
Known effects are the same as under oral Anadrol: rapid mass gain (often 5-10kg in 4-6 weeks), pronounced water retention, paradoxical estrogen effects (gynecomastia research findings possible), hypertension, unfavorable lipid profile, HPG suppression. Liver effects measurably lower than oral.
In recreational research practice Anadrol Oil is often used as alternative for longer Anadrol cycles where the oral form would be limited by hepatotoxicity.
Oxymetholone is on the WADA banned list.
You can order Anadrol Oil as an oily injection solution in 10-pack at 50mg/ml.
This information is for research and educational purposes only. No medical recommendations.
Risks & Safety
Anadrol Oil bypasses the intense liver first-pass of the oral form - but the liver burden reduced in the available data does not change the fact that Oxymetholone is 17-alpha-methylated and ranks among the most potent anabolics of the class.
Documented in studies and the application literature:
- Hepatotoxicity: oral Anadrol is among the most hepatotoxic anabolics overall, the injection form measurably reduces the liver burden but remains hepatotoxic due to the 17-alpha-methyl group.
- Paradoxical estrogen effects: Oxymetholone does not aromatize but directly activates the estrogen receptor. Pronounced water retention and gynecomastia research findings are documented, which a classical aromatase inhibitor does not reliably address.
- Erythropoiesis: Anadrol was originally an anemia medication, a marked hematocrit rise with elevated thrombosis risk is documented.
- Suppression of endogenous production and cardiovascular: a pronounced HPG suppression, hypertension and an unfavorable lipid profile are documented.
Baseline bloodwork before, during and after a research protocol is strongly advised. This classification does not replace medical advice. Risk and responsibility for any actual use lie entirely with the buyer.
Studies
- Double-blind, randomized, placebo-controlled phase III trial of oxymetholone for the treatment of HIV wasting - AIDS 2003, Hengge et al: phase III trial of oxymetholone in HIV wasting.
- Review of oxymetholone: a 17α-alkylated anabolic-androgenic steroid - Clinical Therapeutics 2001, Pavlatos et al: comprehensive pharmacology and safety review.
- Oxymetholone treatment for the anemia of bone marrow failure - Blood 1972, Alexanian et al: classical anemia indication and erythropoiesis data.
- Oxymetholone hepatotoxicity enhanced by concomitant use of cyclosporin A in a bone marrow transplant patient - Clinical and Laboratory Haematology 1994, Wood et al: case report on liver toxicity.
- Trial of an anabolic steroid (oxymetholone) in atherosclerosis with hyperlipoproteinaemia - Atherosclerosis 1973, Barton et al: lipid profile effects under oxymetholone.
